Swine Breeding and Production in Malaysia
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eradicated from Malaysia, and occurrence of CSF can often be attributed to the breakdown of vaccination
programs. Maternal antibodies react with vaccines at the first dose during weaning and second dose at three or
five months which is able to provide lasting immunity is not a common practice in Malaysia.
At the grower to finisher stage, Porcine Respiratory Disease Complex (PRDC) affects growers more as this is
tightly associated with the longer incubation period of the disease after which they get infected in the farrowing
crate. This PRDC is commonly seen in growers to finishers and the aetiology revolves around Mycoplasma
hyopneumonia, M. haemolytica, Pasteurella multocida, Bordetella bronchispetica, Actinobacillus
pleuropneumonia (APP), Porcine Reproductive and Respiratory Syndrome virus(PRRS). PRDC predisposes
these growers to secondary infections which reduce growth performance. Clinical signs are commonly coughing
(either wet and productive or dry and persistent coughs) and sneezing with nasal discharge and gradual
reduction of weight. This is still a persistent scenario even in a vaccinated herd. The severity of the disease is
often only evident at post mortem. Mycoplasma sp. associated cases can be distinguished by persistent dry
cough with non- uniform herd size. The cranio-ventral lung lobes are normally atelectic with marked airways
lymphoid hyperplasia. Serology for antibody titers is able to give an idea of herd infection which will almost
always be positive as it is an endemic disease in most herds. For diagnosis, Fluorescence antibody test (FAT)
and Immunochemistry (IHC) can be applied to lung tissue, whilst Polymerase chain reaction (PCR) and
bacterial culture can be done on airway swab. (Too, 1997) In the case of Pasteurella multocida, wet coughs and
sneezing will be appreciated. Upon post mortem, grayish fibrinous pneumonia is commonly seen carniaventrally
on the lung lobes and variable pleurisy and adhesions in that area. Culture of Pasteurella multocida can give an
idea of infection, but Pasteurella multocida can also be a secondary pathogen. (Too, 1997) APP has two forms,
the acute and chronic forms. In acute cases, growers will have high morbidity and high mortality. Common
clinical signs in acute cases are high temperatures, dyspnoeic, mouth breathing and perhaps bloody foam from
nose. Whereas in chronic cases, growers will have a chronic cough and be ill thirfted. APP can be distinguished
from the other PRDC aetiologies by observing the lesions at post mortems and by culture lung lesions to isolate
the agent. Most commonly, firm and necrotic pneumonia will be seen on the diaphragmatic lung lobes. Pleural
adhesion can be seen in chronic cases. For prevention, carriers should be identified and removed from the herd.
This can be done by serology testing by Enzyme- linked Immunosorbent assay (ELISA) and Complement
Fixation Test (CFT) tests.
At the farrowing stage, Mastitis-Metritis-Agalactia (MMA) seems common amongst Malaysian pig farms,
typically occurring 12 hours to 3 days after parturition. Predisposing factors are unhygienic farrowing crate
causing coliform infection in the mammary glands, decreased water availability, high nutrient density rations,
drastic dietary changes in late gestation, high dietary protein and energy content with inadequate fiber content,
overfeeding, underfeeding, vitamin E and selenium deficiencies. Typical clinical signs in sows are depression
followed by inappetance, restlessness and constipation. Sow will be always on sternum, not allowing piglets to
suckle. On inspection, udder appears slightly enlarged. Initially piglets will be noisy, crying in front of the sow’s
head, running around sow and exploring udder for milk, but later, the piglets will lose interest in the udder, and
will start drinking water or urine from the floor. Piglets will be weak and emaciated with concurrent diseases
and will more prone to be crushed by the sow. Commonly used treatment is oxytocin (30-50units)
intramuscularly repeated every three to four hours. Coliform mastitis cases are given broad spectrum antibiotics
and Flunixin meglumine (Prostaglandin synthetase inhibitor) is used in mammary odema cases. Excitement of
the sow is minimized as much as possible as the release of adrenaline from the excitement phase will block the
effect of oxytocin. Tranquilizer is to be used only on hyper-excitable gilts whereby the hypogalactia is caused
by the adrenaline induced inhibition of milk letdown.
In Malaysia, Thin Sow Syndrome is also a common phenomenon which typically occurs in young sows in the
first lactation. These sows fail to gain weight after the litter is weaned. Predisposing factors are poor nutrition,
heat stress, poor ventilation due to poor farrowing facilities, causing the loss of appetite of lactating sows. These